Here are some actual studies. I think on an individual basis, it should be investigated instead of solely relying on the basic information that appears in the media.
To our knowledge, this is the first study to highlight the presence of pesticides-associated genetically modified foods in maternal,fetal and nonpregnant women’s blood. 3-MPPA and Cry1Ab toxin are clearly detectable and appear to cross the placenta to the fetus. Given the potential toxicity of these environmental pollutants and the fragility of the fetus, more studies are needed, particularly those using the placental transfer approach. Thus, our present results will provide baseline data for future studies exploring a new area of research relating to nutrition, toxicology and reproduction in women. Today, obstetric-gynecological disorders that are associated with environmental chemicals are not known. This may involve perinatal complications(i.e.abortion,pre-maturity, intrauterine growth restriction and preeclampsia) and
reproductive disorders (i.e. infertility, endometriosis and gynecological cancer). Thus, knowing the actual PAGMF concentrations in humans constitutes a cornerstone in the advancement of research in this area.
www.uclm.es/Actividades/repositorio/pdf/doc_3721_4666.pdf
Glyphosate is an active ingredient of the most widely used herbicide and it is believed to be less toxic than other pesticides. However, several recent studies showed its potential adverse health effects to humans as it may be an endocrine disruptor. This study focuses on the effects of pure glyphosate on estrogen receptors (ERs) mediated transcriptional activity and their expressions. Glyphosate exerted proliferative effects only in human hormone-dependent breast cancer, T47D cells, but not in hormone-independent breast cancer, MDA-MB231 cells, at 10⁻¹² to 10⁻⁶M in estrogen withdrawal condition. The proliferative concentrations of glyphosate that induced the activation of estrogen response element (ERE) transcription activity were 5-13 fold of control in T47D-KBluc cells and this activation was inhibited by an estrogen antagonist, ICI 182780, indicating that the estrogenic activity of glyphosate was mediated via ERs. Furthermore, glyphosate also altered both ERα and β expression. These results indicated that low and environmentally relevant concentrations of glyphosate possessed estrogenic activity. Glyphosate-based herbicides are widely used for soybean cultivation, and our results also found that there was an additive estrogenic effect between glyphosate and genistein, a phytoestrogen in soybeans. However, these additive effects of glyphosate contamination in soybeans need further animal study.
Glyphosate is routinely used on GMO crops, as they were specifically created to withstand this pesticide. Non-GMO crops cannot survive Glyphosate.
www.ncbi.nlm.nih.gov/pubmed/23756170
The broad spectrum herbicide glyphosate is widely used in agriculture worldwide. There has been ongoing controversy regarding the possible adverse effects of glyphosate on the environment and on human health. Reports of neural defects and craniofacial malformations from regions where glyphosate-based herbicides (GBH) are used led us to undertake an embryological approach to explore the effects of low doses of glyphosate in development. Xenopus laevis embryos were incubated with 1/5000 dilutions of a commercial GBH. The treated embryos were highly abnormal with marked alterations in cephalic and neural crest development and shortening of the anterior−posterior (A-P) axis. Alterations on neural crest markers were later correlated with deformities in the cranial cartilages at tadpole stages. Embryos injected with pure glyphosate showed very similar phenotypes. Moreover, GBH produced similar effects in chicken embryos, showing a gradual loss of rhombomere domains, reduction of the optic vesicles, and microcephaly. This suggests that glyphosate itself was responsible for the phenotypes observed, rather than a surfactant or other component of the commercial formulation. A reporter gene assay revealed that GBH treatment increased endogenous retinoic acid (RA) activity in Xenopus embryos and cotreatment with a RA antagonist rescued the teratogenic effects of the GBH. Therefore, we conclude that the phenotypes produced by GBH are mainly a consequence of the increase of endogenous retinoid activity. This is consistent with the decrease of Sonic hedgehog (Shh) signaling from the embryonic dorsal midline, with the inhibition of otx2 expression and with the disruption of cephalic neural crest development. The direct effect of glyphosate on early mechanisms of morphogenesis in vertebrate embryos opens concerns about the clinical findings from human offspring in populations exposed to GBH in agricultural fields.
pubs.acs.org/doi/abs/10.1021/tx1001749
Glyphosate, the active ingredient in Roundup®, is the most popular herbicide used worldwide. The industry asserts it is minimally toxic to humans, but here we argue otherwise. Residues are found in the main foods of the Western diet, comprised primarily of sugar, corn, soy and wheat. Glyphosate's inhibition of cytochrome P450 (CYP) enzymes is an overlooked component of its toxicity to mammals. CYP enzymes play crucial roles in biology, one of which is to detoxify xenobiotics. Thus, glyphosate enhances the damaging effects of other food borne chemical residues and environmental toxins. Negative impact on the body is insidious and manifests slowly over time as inflammation damages cellular systems throughout the body. Here, we show how interference with CYP enzymes acts synergistically with disruption of the biosynthesis of aromatic amino acids by gut bacteria, as well as impairment in serum sulfate transport. Consequences are most of the diseases and conditions associated with a Western diet, which include gastrointestinal disorders, obesity, diabetes, heart disease, depression, autism, infertility, cancer and Alzheimer’s disease. We explain the documented effects of glyphosate and its ability to induce disease, and we show that glyphosate is the “textbook example” of exogenous semiotic entropy: the disruption of homeostasis by environmental toxins.
www.mdpi.com/1099-4300/15/4/1416
Glyphosate residue could reach humans and animals through
feed and excreted in urine. Presence of glyphosate in urine and its
accumulation in animal tissues is alarming even at low concentrations.
Unknown impacts of glyphosate on human and animal health warrants
further investigations of glyphosate residues in vertebrates and other
non-target organisms.
www.omicsonline.org/open-access/detection-of-glyphosate-residues-in-animals-and-humans-2161-0525.1000210.pdf
www.sciencedirect.com/science/article/pii/S0278691514002002
The risk assessment of genetically modified (GM) crops for human nutrition and health has not been systematic. Evaluations for each GM crop or trait have been conducted using different feeding periods, animal models, and parameters. The most common result is that GM and conventional sources induce similar nutritional performance and growth in animals. However, adverse microscopic and molecular effects of some GM foods in different organs or tissues have been reported. Diversity among the methods and results of the risk assessments reflects the complexity of the subject. While there are currently no standardized methods to evaluate the safety of GM foods, attempts towards harmonization are on the way. More scientific effort is necessary in order to build confidence in the evaluation and acceptance of GM foods.
www.ncbi.nlm.nih.gov/pubmed/19146501
Genetically modified foods such as soy and corn may be responsible for a number of gluten-related maladies including intestinal disorders now plaguing 18 million Americans, according to a new report released on Tuesday. The report was released by the Institute for Responsible Technology (IRT), and cites authoritative data from the US Department of Agriculture, US Environmental Protection Agency records, medical journal reviews as well as international research.
www.rt.com/usa/gmo-gluten-sensitivity-trigger-343/
The controversy about the health safety of GM foods is complex and good science and its communication are required in order to find solutions.The current guidelines for the safety assessment of GM foods have broad evaluation criterions but no detailed methodologies for testing
safety or thorough guidelines. In order to prove the safety of transgenic foods, it is necessary to exhaust the available possibilities, not discard the previous studies. The advantages of transgenic foods could provide solutions for many problems, but it is first necessary to prove that these foods will not cause other problems. Although numerous advancements can improve the reliability of GM food safety assessment, additional research in other important areas are needed
in order to develop new and more effective methods. Advances in molecular biology, toxicology, biochemistry, and nutrition hold the promise of providing sets of genes and methodologies that serve as biomarkers for a cell’s responses to toxins, allergens, or other compounds. They
will facilitate the development of new tools to facilitate the advancement and assessment of GM crops. The scientific priority is to contribute to the improvement of human and animal health or natural resource management without compromising public safety. More scientific effort and investigation is needed to ensure that consumption of GM foods is not likely to provoke any
form of health problem. The next step in GM crop safety assessment is to have regulatory agencies adopt the developments and recommendations that have been made by advisory committees convened by regulatory agencies and science organizations and put forth in scientific publications.
static.aboca.com/www.aboca.com/files/attach/news/risk_assessment_of_genetically_modified_crops_for_nutrition.pdf 
